简介：Asoneoftheidealtoolsformonitoringtheformationandevolutionofsolarstorms,thevacuumultraviolet(VUV)detectorshouldhavebothafasttemporalresponseandanarraystructurethatenablesimageformation.Here,bycombininganontraditionalgrapheneprocessingtechniquewithtraditionalmetalorganicchemicalvapordepositionepitaxytechnology,wecreatedhybridheterostructure(HH)arraysofp-Gr/AlN/p-SiwithVUVphotovoltaicresponsecapabilityandsiliconintegrationpotential.TheHHarraysnotonlyexhibitultrafasttemporalresponse(risetimeofonly120ns)andanextremelyhighIon∕Ioffratioof107,butalsoachievetheimagingdemonstrationofaVUVpatternforthefirsttime.TheHHtechniqueprovidesapossiblenewpathforthedevelopmentofVUVimagingdevices.

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简介：Whenasilicaopticalfiberisusedfortransmittingultraviolet(UV)light,theproblemsassociatedwiththedamageandtransmissiondeteriorationofsilicafiberintheUVregionneedtobesolvedinthefabricationprocess.Inthispaper,animprovedsilicafiberfabricationprocesswaspresentedtoenhanceUVtransmission,whichcombinedtheUVpre-irradiationonhigh-OHconcentrationpreformwithheattreatmentwhiledrawingfiber.Theimpactofthestructuraldefectsandcolorcenterswasdiminishedgreatlybythenewmethodoffabrication.Theexperimentalresultsaccordedwiththetheoreticalanalysis,andtransmissionwasimprovedabout0.4-0.7dB/mforwavelengthfrom350to400nm.

简介：紫外(紫外)放射穿上重要效果生态系统，环境，和人的健康，以及大气的过程和气候变化。二紫外放射数据集在这份报纸被描述。一个人时时包含从2005～2015在40个中国生态系统研究网络车站测量的紫外放射的观察。CUV3宽带辐射计被用来观察紫外放射，与5%的精确性，它满足世界气象学组织测量标准。极值方法被用来控制测量数据集的质量。另外的数据集包含用与一个混合模型相结合的一个所有天空评价模型是计算的每天累积的紫外放射估计。从1961～2014的重建的每天紫外的放射数据跨度。吝啬的绝对偏爱错误和root-mean-square错误至多比30%小吝啬的偏爱错误价值的车站，和大多数是否定的，它显示紫外放射紧张的低估。这些数据集能在紫外放射改进我们空间、时间的变化的基本知识。另外，这些数据集能在潜力的研究被使用臭氧形成和大气的氧化，以及生态的进程的模拟。

简介：Electrophoresis,chromatography,immunoassay,sequencingandothertimeconsumingap-proacheshavebeendevelopedtodetermineDNAbasemismatching,oxidativelesionorstrandbreaks.Sometimes,however,onlyqualitativeinformationisenoughtodecidewhethermutationhashappenedtoDNAanditsextent.Convolutionspectrometry(CS),anewtechniquetodiscoverultrafmedifferenceonultraviolet(UV)absorptionofdifferentsubstances,isoriginallyemployedtofindoutanysubtlemutationofDNAinducedbyUVradiation.Muta-tiveDNAiscomparedwithegocriteriabasedonthespectraoftheformerDNA,anydifferenceisquantitativelyex-pressedbydispersion(5).Visiblechangescannotbeobservedonsecond-derivativespectrauntilthemutationgets5upto11.48%.DimethylsulfoxideisanintensifierofUV254nminducedDNAmutationandprotectorat365nm,whichissimplyconfirmedbyincreasinganddecreasing5.Everyconvolutionproceduretakeslessthan1min.Convolutionspectrometryprovidesafast,simple,sensitiveandinexpensivealternativetodetermineDNAmutation,andtoscreenanti-mutationalmedicines.

简介：ThepumpedsystemoftheXeC1excimerlaserhasbeendesignedandappliedtoanalyzeUVXeCllaserspectroscopycharacteristics.UndertheproportionofmixedgasofHCl:Xe:He=0.1%:1%:98.9%,excimerlaserlightwasgeneratedbystableglowdischargeprocess.Thelaserspectrum,pulseduration,andlaserpowerpropertieswereobtained.TheresultshowsthatthisXeClexcimerlaserexhibitsuniquespectralproperties,withtwopeaksbetween307.7nmand308.5nmathighpressurewithapumpedpowerof1.3959MW/cm~3.ThetransitionreliesonthestrongesttransitionsbetweenB-XandC-X.Themaximum-intensitytransitionofspectroscopyisBtoXenergylevels.Thelaserparametersareasfollows:minimaldurationof15.42ns,arepetitionratefrom0.5Hzto5Hz,singlepulsestablepowerof400mJ,andbeamdivergenceangleof3mrad.ThelasercanbeusedtostudyUVspectroscopy,laserablationsamplingandsputteredpinnateform.

简介：Anegativeunipolarpulsedvoltageisappliedtostudyinternalelectricalparametersofthexenonfilleddielectricbarrierdischarge（DBD）sources.TheVUVradiationemittedfromthesesourcesischaracterizedbymeansofthephotoluminescenceintensityoftheredphosphorpellet.TheredphosphorconvertstheVUVradiationintovisibleradiationandtheemissionspectraincludeapeakat619.56nm.Theemissioncharacteristicsoftheredphosphorareanalyzedintermsofthepressure-distance（pd）,risetimeandfrequencyofthepulsedvoltagewaveform.Theemissionintensitymeasuredatdifferentoperationalconditionsconfirmsthattheformationanddecayofthexenonexcimer,Xe2*,increasewiththeincreaseinreducedelectricfield,E/N.AfterexceedingcertainlimitsofE/N,theintensityofXe2*decreasesrapidly.

简介：在第二等的自河的推迟的固体的集中经常广泛地变化，导致满足消毒标准的紫外剂量的经常的调整。Inaddition，需要的消毒率是困难的有时完成。作者学习了颗粒分布，联系粒子的烘便的大肠菌(F.C)的贡献，和他们对紫外消毒的影响。一个联合消毒过程(有随后的UVdisinfection的氯化)被测试改进消毒效果。结果显示推迟的固体的内容，特别大粒子，在紫外消毒效率上有强壮的影响；D>10个μm粒子联系了F.C。是困难的被消毒并且是在激活曲线的F.C的跟踪的主要部分。氯化前能减少在第二等的自河和变换的粒子的数字大粒子进小的，在紫外消毒上减少粒子的影响并且提高联合过程的抵抗能力到粒子装载。

简介：Energylevels,radiativerates,oscillatorstrengthsandlinestrengthsarereportedfortransitionsamongthelowest97levelsofthe(1s22s22p6)3s23p2,3s23p3d,3s3p3,3p4,3s3p23d,and3s23d2configurationsofRbXXIV.AmulticonfigurationDirac–Fock(MCDF)methodisadoptedforthecalculations.Radiativerates,oscillatorstrengths,andlinestrengthsareprovidedforallelectricdipole(E1),magneticdipole(M1),electricquadrupole(E2),andmagneticquadrupole(M2)transitionsfromthegroundleveltoall97levels,althoughcalculationsareperformedforamuchlargernumberoflevels.Toachievetheaccuracyofthedata,comparisonsareprovidedwithsimilardataobtainedfromtheFlexibleAtomicCode(FAC)andalsowiththeavailabletheoreticalandexperimentalresults.Ourenergylevelsarefoundtobeaccuratetobetterthan1.2%.WavelengthscalculatedarefoundtolieinEUV(extremeultraviolet)andx-rayregions.Additionally,lifetimesforall97levelsareobtainedforthefirsttime.

简介：Tostudytheroom-temperaturestabledefectsinducedbyelectronirradiation,commercialborosilicateglasseswereirradiatedby1.2MeVelectronsandthenultraviolet(UV)opticalabsorption(OA)spectraweremeasured.Twocharacteristicbandswererevealedbeforeirradiation,andtheywereattributedtosilicondanglingbond(E’-center)andFe3+species,respectively.TheexistenceofFe3+wasconfirmedbyelectronparamagneticresonance(EPR)measurements.Afterirradiation,theabsorptionspectrarevealedirradiation-inducedchanges,whilethecontentofE’-centerdidnotchangeinthedeepultraviolet(DUV)region.TheslightlyreducedOAspectraat4.9eVwassupposedtotransformFe3+speciestoFe2+speciesandthistransformationleadstotheappearanceof4.3eVOAband.Bycalculatingintensityvariation,thetransformationofFewasestimatedtobeabout5%andtheopticalabsorptioncrosssectionofFe2+speciesiscalculatedtobe2.2timeslargerthanthatofFe3+species.Peroxylinkage(POL,≡Si–O–O–Si≡),whichresultsina3.7eVOAband,isspeculatednottobefromSi–ObondbreakbutfromSi–O–Bbond,Si–O–Albond,orSi–O–Nabondbreak.Theco-presencedefectwithPOLisprobablyresponsiblefor2.9-eVOAband.

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简介：Three—layerback—propagationneuralnetworkswereusedforprocessingtheinformationprovidedbytheultravioletspectraoffive—componentmixtures,affectingthesimultaneousdeterminationofthecomponentswithsatisfactoryresults.

简介：Toinvestigatetheprotectiveeffectofepigallocatechingallate(EGCG)ontheimmunefunc-tionofdendriticcells(DCs)afterultravioletBirradiation(UVB)anditsunderlyingmechanisms,themonocyteswereisolatedfromperipheralbloodandcultivatedintoDCswithcytokines,suchasGM-CSFandIL-4.DCswereharvestedaftercultivationfor7dandsubjectedtoirradiationwithdifferentdosagesofUVB.Then,200μg/mlofEGCGwereaddedincertaingroupsimmediatelyafterirradiation.DCssimplytreatedwithUVBortreatedwithbothUVBandEGCGwereco-culturedwithlymphocytes,andMTFassaywasusedtodetecttheabilityofDCstostimulateproliferationoflymphocytes.SurfacemarkersCDS0,CD86,HLA-DRandCD40weredetectedbyflowcytometry,andthelevelsofIL-10andIL-12secretedfromDCs24haftercultivationweremeasuredbyELISA.ItwasdemonstratedthatUVBirradiationcouldinhibittheabilityofDCstostimulatetheproliferationoflymphocytesandsurfaceexpressionsofCDS0,CD86,HLA-DRandCD4onDCsinadose-dependentmanner.TheinhibitionrateofDCswasimprovedtosomeextentaftertreatmentwith200μg/mlofEGCG.Whentheconcentra-tionofEGCGexceeded100μg/ml,theenhancingeffectofEGCGontheexpressionoftheco-stimulat-ingmoleculesonDCscouldbedemonstratedinadose-dependentmanner.UVBshowednosignificantinfluenceonthesecretionofIL-10andIL-12fromDCs,whileEGCGcoulddown-regulatethesecretionlevelofIL-12andup-regulatethatofIL-10.ItisconcludedthatEGCGcanantagonizetheinhibitoryeffectonDCsinducedbyUVBirradiation.Thisfunctionhassomerelationshipwithitsprotectingeffectoftheexpressionoftheco-stimulatingmoleculeonthesurfaceofDCsandthesecretionlevelofIL-10andIL-12.

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简介：AbstractObjective:The plant polyphenol resveratrol (3,4′,5-trihydroxystilbene) (RSV) has been proposed for use because of its protective effect on ultraviolet (UV)-induced skin disorders. In UVB-induced skin damage, cell autophagy and apoptosis have been approved to prevent the damage and to contribute to the cytoprotective role of RSV; however, the detailed mechanism remains unknown. So, we conducted this study to investigate the cytoprotective effects of RSV on UVB-irradiated human epidermal keratinocytes (HEKs) and its undergoing mechanisms.Methods:Secretion of thirty-six inflammatory cytokines of HEKs induced by 50 mJ/cm2 UVB at 0, 12, 24, and 48 hours were detected by a human cytokine assay and the interleukin (IL)-8 protein level in the culture media were determined by ELISA. Next, HEKs were treated with or without 100 μmol/L RSV in the presence or absence of 50 mJ/cm2 UVB, and activator protein 1 and NF-κB-related proteins were measured by Western blot. Furthermore, cells exposed to UVB radiation were treated with apoptosis activators procaspase-activating compound 1 (PAC-1), apoptosis activator 2 (AA2) or RSV to investigate the effect of RSV on the percentage of apoptotic cells by flow cytometry. Then cells were treated with autophagy inhibitors LY294002, 3-methyladenine (3-MA) or RSV in the presence of UVB and chloroquine (CQ) to investigate the effect of RSV on autophagy through detecting microtubule-associated protein 1 light chain 3 (LC3) expression by western blot. Finally, the effect of LY294002, 3-MA, ATG5 siRNA, PAC-1, and AA2 on RSV-mediated reduction of IL-8 expression was determined by ELISA assay.Results:RSV treatment decreased the secretion of IL-8 (UVB vs. UVB+ RSV: [1454.05 pg/mL ± 52.95 pg/mL] vs. [553.68 pg/mL ± 206.03 pg/mL], P < 0.001), and downregulated the protein level of c-Fos in UVB-irradiated HEKs (UVB vs. UVB+ RSV: [0.103 ± 0.009] vs. [0.048 ± 0.015], P < 0.01). In UVB-irradiated HEKs, the result of western blot showed that LY294002 and 3-MA inhibited RSV-induced LC3 II accumulation (UVB + CQ + RSV vs. UVB + CQ + 3-MA+ RSV vs. UVB + CQ + LY294002+ RSV: [1.15 ± 0.03] vs. [0.77 ± 0.13] vs. [0.67 ± 0.13], P < 0.01), and the result of flow cytometry showed that PAC-1 and AA2 prevented RSV from reducing cell apoptosis (UVB+ RSV vs. UVB+ PAC-1 + RSV vs. UVB+ AA2+ RSV: [19.56% ± 0.62%] vs. [94.33% ± 0.15%] vs. [94.97% ± 1.91%], P < 0.001). The data of ELSA assay showed that LY294002, 3-MA, and ATG5 siRNA reversed the RSV-mediated inhibition of IL-8 protein secretion by UVB-irradiated HEKs (UVB + LY294002 vs. UVB + LY294002 + RSV: [3283.00 pg/mL ± 444.05 pg/mL] vs. [1608.58 pg/mL ± 128.42 pg/mL], P < 0.05; UVB + 3-MA vs. UVB+ 3-MA+ RSV: [2941.88 pg/mL ± 103.80 pg/mL] vs. [1867.51 pg/mL ± 153.84 pg/mL], P < 0.01; UVB+ siATG5 vs. UVB+ siATG5+ RSV: [2530.11 pg/mL ± 685.34 pg/mL] vs. [3011.42 pg/mL ± 435.69 pg/mL], P > 0.05), whereas neither PAC-1 nor AA2 exerted similar effects.Conclusion:RSV may regulate autophagic flux to inhibit IL-8 expression in UVB-challenged keratinocytes.

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