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简介：ThispaperstudiesRabinovitch'scompressionexperimentsongraniteandchalkandproposesanoscillatingdipolemodeltoanalyseandsimulatetheelectromagneticradiationphenomenoncausedbyfractureofrocks.Ourmodelassumesthattheelectromagneticradiationpulsesareinitiatedbyvibrationsofthechargedrockgrainsonthetipsofthecrack.Thevibrationsoftherockgrainsarestimulatedbythepulsesofthecracks.OursimulationsshowcomparableresultswithRabinovitch'scompressionexperiments.Fromthesimulationresults,itverifiesanassumptionthatthecrackwidthisinverselyproportionaltothecircularfrequencyelectromagneticradiation,whichispresentedbyRabinovitchetal.Thesimulationresultsalsoimplythat,byusingouroscillatingdipolemodeltogetherwithRabinovitch'stwoequationsaboutthecracklengthandcrackwidth,wecanquantitativelyanalyseandsimulatetheelectromagneticradiationphenomenon,whichisinducedfromthefractureoftherocks.

简介：当一个中等稳定的阶段在内部反应期间外面被猛抛时，在散开地区以内的渗透的原子的行为能基于热力学的考虑被解释。在Ni-Cr合金at1125℃的内部nitridation期间向降水前面通过矩阵为“登高”证实渗透的种类的散开，6000块N_2压力被预言。氮的如此的行为对在瓦格纳的边界条件相反“对内部反应的s描述。与降水反应联系的一个体积变化导致了在合金表面和internalnitridation前面之间的一个压力坡度。应力消除主要由镍的运输发生了到气体/金属的接口。管子控制散开creep在nitriding期间是主导的压力住所机制冲淡在在流动NH_3+H_2煤气的混合物下面的700℃的Ni-Cr合金。

简介：AbstractBackground:Osteoarthritis (ΟΑ) is characterized by cartilage breakdown and subchondral sclerosis. Micro-fractures of the calcified tissues have been, also, detected, but their exact role has not been elucidated yet. This study was to examine the frequency of cracks during OA progression and to correlate them with the underlying cellular modifications and matrix metalloproteinase-2 (MMP-2) expression using histological/immunohistological methods.Methods:Overall, 20 patients and 3 controls (9 specimens per patient), aged 60-89 years, diagnosed with hip/knee OA were included. The development of cracks was examined in 138 sections, whereas the expression of MMP-2 was examined in 69 additional sections.Results:Based on Mankin score, three groups of OA severity were analyzed: Group I (mild) was constituted of sections with score 1-5 while Groups II (moderate) and III (severe) with score 6-7 and greater or equal to 8, respectively. Demographic characteristics did not reveal any association between the number of microdefects and age or body mass index (BMI). Cartilage micro-cracks were increased during moderate and severe OA, while bone cracks were increased during mild and severe OA. In knee OA, cartilage cracks were not correlated with Mankin score, whereas in hip OA they appeared association with severity score. Bone cracks were positively correlated with matrix apoptotic osteocytes and osteoblastic cells, but not with osteoclasts. MMP-2 immunostaining was increasing by OA severity in the osteochondral unit. Similarly, MMP-2 was expressed on the microcracks’ wall mainly in Group III.Conclusion:Our data displayed that bone cracks during primary OA stages, represent an early adaptative mechanism aiming to maintain cartilage integrity. Accumulation of bone defects and concomitant increase of apoptotic osteocytes activated an abnormal remodeling due to osteoblastic activity, in which MMP-2 played a pivotal role, leading to subchondral sclerosis promoting further osteochondral deformities.